Friday, December 01, 2006

Researcher, Pro-Life Advocates React to Abortion Drug-Breast Cancer Study

A leading biochemist who has published internationally-respected studies on breast cancer and a top pro-life organization both reacted to a study released Thursday showing the RU 486 abortion drug helped suppress the breast cancer gene in mice. They agreed the drug will not likely benefit humans. The University of California study showed that the RU 486 abortion drug works to suppress the progesterone hormone that triggers the breast cancer gene to produce cancerous tumors. But Dr. Joel Brind, a Yale-educated endocrinologist who teaches biology at Baruch College in New York, told LifeNews.com he doesn't think the drug will offer any benefit to women. "There is reason to believe that RU 486 as an anti-progestin might suppress growth of breast cancer in an experimental system in rodents, but RU 486 also neutralizes the essential hormone cortisol which mice do not make," Brind said. "Therefore, there is good reason to believe that such a drug would not be an effective anti-cancer drug in people," he told LifeNews.com. --Click the title of this post to read the full article from its source--

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Study: Abortion pill blocks breast cancer gene

American scientists caution that their research is still in the exploratory stages, but recent results have shown the abortion drug RU-486 prevented tumors in mice bred with a breast cancer gene. Although no one is suggesting women use the abortion pill to prevent breast cancer, the experiment did show that RU-486 blocks a hormone called progesterone, which activates the breast cancer gene BRCA1. "All of us have to be cautious," said cell biologist Eva Lee of the University of California, Irvine, who led the research published in Friday's edition of the journal Science. "But I do think if there is a better anti-progesterone available, hopefully there will be other options in the future for these women." Women today have few options to prevent breast cancer, and if researchers could produce a safer hormone blocker it would offer a viable alternative for women with the BRCA1 gene.Cancer specialists not involved with the experiment praised the work, even as they warned women not to get their hopes up. --Click the title of this post to read the full article from its source--

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